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Host Immune Responses and Pathogenesis to Brucella spp. Infection
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Brucellosis, caused by the facultative intracellular bacteria Brucella species, is one the most prevalent zoonosis worldwide. • The articles described in this book report several aspects of host-Brucella interactions. • The findings described here will help to advance in the comprehension of bacterial pathogenesis and contribute to the future development of drugs or vaccines to control brucellosis.
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Keywords
- (p)ppGpp
- adhesins
- ASC
- brain microvascular endothelial cells
- Brucella
- Brucella abortus
- Brucella abortus 544
- brucellosis
- canonical inflammasome
- caspase-1
- caspase-11
- Cell Cycle
- Chemokines
- chronic infection
- decidualization
- divalent vaccine
- endothelial cells
- Extracellular matrix
- fibronectin
- gDNA
- HSC
- human endometrial cells
- Ig-like domain
- IL-10
- IL-1β
- internalization
- intracellular replication
- L7/L12
- Macrophages
- MHC
- monomeric autotransporters
- n/a
- neurobrucellosis
- NLR
- non-canonical inflammasome
- Omp25
- oral infection
- Persistence
- platelets
- polar localization
- pyroptosis
- Recombinant vaccine
- Reference, information & interdisciplinary subjects
- replication niche
- Research & information: general
- reservoir
- rsh
- ST2 receptor
- Survival
- thema EDItEUR::G Reference, Information and Interdisciplinary subjects::GP Research and information: general
- trimeric autotransporters
- Vaccine candidates
- Virulence Factors
Links
DOI: 10.3390/books978-3-0365-2419-1Editions