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Cell Death, Inflammation and Oxidative Stress in Neurodegenerative Diseases

Cell Death, Inflammation and Oxidative Stress in Neurodegenerative Diseases

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Neurogenerative diseases encompass very different pathologies, which can be demyelinating or nondemyelinating, but which have common mechanisms such as cell death, oxidative stress and inflammation. A better understanding of these mechanisms allows the search for biomarkers and targets for new therapies. This special issue brings together different data on Alzheimer's disease, Parkinson's disease, and multiple sclerosis, detailing the mechanisms of cell death (necroptosis, ferroptosis), oxidative stress and inflammation but also the possibilities of neuroprotection via 5 research articles and 6 review articles. The different reviews allow us to take stock of cell death, oxidative stress and neuroinflammation in the context of neurodegenerative diseases but also in relation to other pathologies where these processes are involved.

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Keywords

  • Alzheimer’s disease
  • Apoptosis
  • Astrocytes
  • blood-brain barrier (BBB)
  • brain injuries
  • brain ischemia
  • breast cystic fluid
  • Cancer
  • catechin
  • Cell Death
  • central nervous system (CNS)
  • clozapine
  • Cytokines
  • Diffuse Axonal Injury
  • Docosahexaenoic acid
  • Endocytosis
  • ferroptosis
  • FLIM
  • geniculate bodies
  • glia
  • glutathione peroxidase 4
  • GSH
  • Huntington’s disease
  • hyperglycemia
  • hyperinsulinemia
  • Inflammation
  • iron metabolism
  • L-α-phosphatidylcholine
  • Leukocytes
  • Macrophages
  • medicine
  • Mice
  • Microglia
  • Monocytes
  • Mood Disorders
  • mPTP
  • n/a
  • Nanomedicine
  • necroptosis
  • necrosis
  • nerve degeneration
  • neurodegeneration
  • neurodegenerative disease
  • Neurodegenerative Diseases
  • neurofilament proteins
  • Neuroglia
  • neuroinflammation
  • Neurons
  • Neuroprotection
  • neuropsychiatric disorders
  • Neutrophils
  • optic tract
  • Oxidative Stress
  • parkinson’s disease
  • phosphatidylserine
  • platelets
  • ROS
  • S100B protein
  • small molecules
  • Stroke
  • superior colliculi
  • synaptophysin
  • system xc−
  • T lymphocytes
  • TDP-43 proteinopathies
  • traumatic
  • type 2 diabetes (T2DM)

Links

DOI: 10.3390/books978-3-0365-2911-0

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