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Molecular and Cellular Advances in Endometriosis Research

Molecular and Cellular Advances in Endometriosis Research

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Endometriosis is defined as the presence of endometrial-like endometrial cells, glands, and stroma outside the uterus, causing a strong inflammatory-like microenvironment in the affected tissue. The exact prevalence of endometriosis is unknown, but estimates range from 2%–10% of women of reproductive age to 50% of infertile women. The etiopathogenesis of endometriosis remains controversial—immune, hormonal, genetic, and epigenetic factors may all be involved, and several theories have been proposed to explain it.This book aims to summarize some of the most recent advancements in basic and translational science (immunology, cell biology, genetics, and epigenetics) in the field of endometriosis that may create new scenarios and change our perspective of the topic.

This book is included in DOAB.

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Keywords

  • Annexin A1
  • BMI-1
  • ceRNA
  • Chemokines
  • Chromatin
  • Chronic Pain
  • circular lncRNA (circRNA)
  • Cytokines
  • Cytoskeleton
  • dapsone
  • DDS-NHOH
  • EMT
  • endometrial cancer
  • endometriosis
  • epigenetics
  • epithelial to mesenchymal transition
  • epithelial tumor cells
  • EZH2
  • glycyrrhetinic acid
  • Human Disease
  • Inflammation
  • inflammation mediators
  • Lipoxin
  • Lipoxin A4
  • lncRNAs
  • long non-coding RNAs
  • long noncoding RNA (lncRNA)
  • long noncoding RNAs
  • medicine
  • microRNA
  • migration
  • molecular basis of endometriosis
  • nerve signalling
  • neurogenic inflammation
  • neuroimmune modulation
  • non-hormonal treatment
  • noncoding RNA (ncRNA)
  • ovarian cancer
  • peripheral nerve
  • peritoneal fluid
  • proliferation
  • receptors
  • red blood cell
  • Resolvin
  • review
  • sponging
  • stroma
  • Th1 cells
  • Th17 cells
  • Th2 cells
  • Treg cells

Links

DOI: 10.3390/books978-3-0365-2802-1

Editions

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