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Molecular Pathways in Cancers

Molecular Pathways in Cancers

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This book includes some recent works providing the readers with novel relevant findings about the main signaling pathways that govern the molecular pathogenesis of some of the highest prevalent human tumors, which are the basis for developing alternative therapeutic strategies to improve patient outcomes.

This book is included in DOAB.

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Keywords

  • acetylation
  • actin cytoskeletal reorganization
  • Adaptive Immunity
  • advanced ovarian cancer
  • anti-receptor therapy
  • BET inhibitors
  • biomarkers
  • bladder cancer initiation
  • Breast cancer
  • CALR
  • Cancer
  • cancer immunology
  • cancer therapeutics
  • carcinogenicity
  • CD99 agonist
  • Clinical & internal medicine
  • complement system
  • Diseases & disorders
  • DUSP1
  • EGFR dimerization
  • Endocytosis
  • epigenetic
  • Epigenome
  • Epithelial Cells
  • FAK dephosphorylation
  • Genome
  • Glioblastoma
  • gp120
  • hepatocellular carcinoma
  • human immunodeficiency virus type 1
  • immune checkpoint inhibitors
  • Immune Evasion
  • Immunotherapy
  • innate immunity
  • JAK2
  • luminal breast cancer
  • MAPK
  • matrix protein p17
  • medicine
  • metastasis
  • metastatic melanoma
  • Methylation
  • migration and invasion
  • Mitochondria
  • molecular targeted agents
  • MPL
  • MS-determined phosphopeptides
  • mTOR
  • myeloproliferative neoplasms
  • n/a
  • Nef
  • NIMA-related kinase 1 (NEK1)
  • non-coding RNA
  • OMD
  • oncogenic signaling pathway
  • oncology
  • ovarian cancer
  • Oxidative Stress
  • OXPHOS
  • pancreatic ductal adenocarcinoma
  • partial EMT
  • patient survival
  • pBRD4
  • PI3K
  • PP2A
  • PRELP
  • prognosis
  • prostate cancer
  • PTPN12
  • Rac1
  • Reactive Oxygen Species
  • Resistance
  • reverse transcriptase
  • RhoA
  • SET
  • signaling pathways
  • small-cell lung cancer
  • snail
  • splicing pathway
  • TAK-228
  • TAT
  • thioridazine (THD)
  • tight junction
  • tousled-like kinase (TLK)
  • TPOR
  • trastuzumab
  • tripeptide
  • triple negative breast cancer
  • triple-negative breast cancer
  • tumor immune microenvironment
  • Tumor microenvironment
  • tumor suppression gene
  • Warburg effect
  • yes-associated protein 1 (YAP1)

Links

DOI: 10.3390/books978-3-0365-3681-1

Editions

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