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State of the Art in Idiopathic Pulmonary Fibrosis
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This reprint is a compilation of ten publications, including original-research and review articles, and the accompanying editorial within the scope of a Special Issue entitled: "State of the Art in Idiopathic Pulmonary Fibrosis" published in Cells.
This book is included in DOAB.
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Keywords
- (pan-)HDAC-inhibitor
- 3D cultures
- adverse event
- airway epithelium
- alveolar epithelial cells
- alveolar epithelial progenitors
- alveolar fibroelastosis
- amphiregulin
- antibody folding
- antifibrotic treatment
- AT2 cells
- basal cells
- bleomycin
- bleomycin-induced pulmonary fibrosis
- body mass index
- bronchial epithelium
- bronchiolar basal cells
- bronchiolization
- CD24
- CK5
- Class I-HDAC-inhibitor
- discontinuation
- Drug Screening
- epidermal growth factor
- epidermal growth factor receptor
- epithelial populations
- ER stress
- ErbB-signaling
- fibroblast-to-myofibroblast differentiation (FMD)
- FK506-binding protein
- Flow Cytometry
- histone acetylation
- histone deacetylase (HDAC)
- idiopathic pulmonary fibrosis
- idiopathic pulmonary fibrosis (IPF)
- IGF1
- IGF1R
- immunophilin
- intermediate epithelial cells
- interstitial fibrosis
- interstitial lung disease
- IPF
- IPF modelling
- Lung
- lung development
- lung fibrosis
- lung transcriptomic profile
- LysoTracker
- medicine
- MUC5B
- n/a
- neuregulin 1
- NGFR
- Nintedanib
- non-histone protein acetylation
- peptidyl-prolyl isomerase
- pirfenidone
- pulmonary fibrosis
- recombinant FGF10
- single cell RNA sequencing
- tacrolimus
- TGF-α
- TGF-β
- transitional states
- type-II alveolar epithelial cell (AECII)
- Tyrosine Kinase Inhibitor
Links
DOI: 10.3390/books978-3-0365-6384-8Editions