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Pediatric Cancers
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Childhood cancer or pediatric cancer is one of the leading causes of death among children. Treatment strategies for pediatric cancers include chemotherapy, surgery, radiation, and stem cell transplantation. These therapeutic regimens are well known to develop highly toxic and long-lasting side-effects including secondary cancers in the future lifespan of pediatric patients. Despite these intensive therapeutic regimens, cancer may still relapse as refractory and metastatic cancer. Therefore, further understanding the causes of different pediatric cancers, mechanisms of drug-resistance, and causes of relapse and metastasis is required for developing and advancing less toxic and more effective therapeutic strategies for pediatric cancers. Recent advancements in immunotherapies and targeted therapeutic approaches using pharmacological inhibitors are one step in this direction.In this Special Issue of Cancers, authors contributed original research and review articles focusing on different aspects of pediatric cancer development, causes, maintenance, and therapeutic strategies. The collected articles in this Special Issue will further enhance our knowledge and understanding of pediatric cancers and drive the development of novel therapeutic strategies.
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Keywords
- 18F-FDG PET/CT
- acute myeloid leukemia
- adjuvant
- AdOx
- allostasis
- ATRA
- B7-H3
- bacteremia
- bioinformatics analysis
- Brain
- c-Myc/N-Myc
- Cancer
- cancer predisposition syndromes
- Cell Differentiation
- cell surface proteomics
- chemotherapy
- chemotherapy response
- childhood acute leukemia
- childhood cancer
- childhood malignant bone tumors
- Children
- chloroma
- Clinical & internal medicine
- clinical malformations
- Cohort study
- combination therapy
- CRISPR-Cas9
- cytotoxic cancer treatment
- DIC
- diffuse intrinsic pontine glioma
- Diseases & disorders
- Drug Discovery
- drug repurposing
- Drug transporters
- DSRCT
- DZNep
- early complication
- early life stress
- ependymoma
- epigenetic
- epigenetics
- EPZ005687
- ERK inhibition
- Ewing sarcoma
- EWSR1-WT1
- EZH2
- EZRIN
- Fimepinostat
- fungal secondary metabolites
- Glioma
- hepatoblastoma
- high-risk
- hyperleukocytosis
- immune checkpoint inhibitor
- immune microenvironment
- Immunotherapy
- in vitro model
- Infections
- INI1
- inoperable
- Interventions
- Ki67
- late effects
- LPA
- LPAR1
- maintenance therapy
- malignant rhabdoid tumor
- MDR
- medicine
- medulloblastoma
- melanotic neuroectodermal tumor of infancy
- metastasis
- metastatic
- metronomic chemotherapy
- molecular group
- myeloid sarcoma
- n/a
- Natural Products
- neoadjuvant
- nephroblastoma
- Neuroblastoma
- oncology
- oncolytic virus
- outcome
- pediatric
- pediatric cancer
- pediatric cancers
- pediatric liver tumors
- PI3K pathway
- Pneumonia
- preclinical model
- prognosis
- prognosis prediction
- radiogenomics
- random forest
- RD
- recurrent
- regulation network
- RH30
- Rhabdomyosarcoma
- risk factor
- risk model
- risk stratification
- RNA-binding proteins
- Sarcoma
- serine-threonine kinase receptor-associated protein
- small cell undifferentiated (SCUD) hepatoblastoma
- SMARCB1
- soil microbes
- solid tumor
- somatic disease
- SOX4
- Stress
- stress dysregulation
- Stress reactivity
- stress regulatory pathways
- survival outcome
- survivorship
- systemic treatment
- targeted therapies
- tumor
- tumor metastasis
- tumor surveillance
- ulixertinib
- Vaccine
- VIPN
Links
DOI: 10.3390/books978-3-0365-9025-7Editions