Explore
Regulation of Autophagy under Stress
0 Ungluers have
Faved this Work
Login to Fave
Autophagy is a conserved degradative mechanism essential for cellular homeostasis in eukaryotic organisms. It is a cellular response mechanism that helps cells adapt and survive under various stressful conditions. It involves the degradation and recycling of cellular components to maintain cellular homeostasis and protect against damage from stressors like nutrient deprivation, oxidative stress, infection, and protein misfolding. However, the understanding of the molecular mechanisms of autophagy regulation is still in significant development, including the involvement of gasotransmitters and small signaling molecules, such as those produced under ROS, RNS and RSS. Dysregulation of autophagy has been implicated in various diseases, making it an important area of research to improve biomedical therapies and strategies for overcoming the challenges from environmental stresses. Therefore, this Special Issue aims to review the current developments regarding autophagy research.
This book is included in DOAB.
Why read this book? Have your say.
You must be logged in to comment.
Rights Information
Are you the author or publisher of this work? If so, you can claim it as yours by registering as an Unglue.it rights holder.Downloads
This work has been downloaded 14 times via unglue.it ebook links.
- 14 - pdf (CC BY) at Unglue.it.
Keywords
- Aging
- Alzheimer
- AMD
- autophagosomes
- Autophagy
- autophagy-related genes (ATG)
- autophagy-related protein
- Biochemical engineering
- Biotechnology
- Brain
- Cerebellum
- CHK2
- Clinical Trial
- colorectal cancer
- degradation
- Dr
- energy stress
- ER stress
- Extracellular matrix
- Glaucoma
- GPCR
- Gq
- Harderian gland
- Harlequin
- Heart
- hydrogen sulfide
- hypoxia
- Inflammation
- intestinal homeostasis
- lipophagy
- Mechanotransduction
- Melatonin
- metabolic syndrome
- Mitochondria
- mitochondrial diseases
- mitophagy
- mTOR
- non-alcoholic fatty liver disease
- nutrient stress
- Oxidative Stress
- OXPHOS
- parkinson
- persulfidation
- PINK1
- PPARα
- rapamycin
- Reactive Oxygen Species
- Régulation
- ROS
- skeletal muscle
- Stevia
- stevioside
- stress response
- Technology, engineering, agriculture
- Technology: general issues
- ULK1
- vacuole
Links
DOI: 10.3390/books978-3-0365-8895-7Editions